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Kamata H, Hirata H (1999) Redox regulation of cellular signaling. Įvan AP, Worcester EM, Coe FL, Williams J Jr, Lingeman JE (2015) Mechanisms of human kidney stone formation. Įvan AP, Coe FL, Lingeman JE, Shao Y, Sommer AJ, Bledsoe SB, Anderson JC, Worcester EM (2007) Mechanism of formation of human calcium oxalate renal stones on Randall’s plaque. Įvan AP, Lingeman JE, Coe FL, Parks JH, Bledsoe SB, Shao Y, Sommer AJ, Paterson RF, Kuo RL, Grynpas M (2003) Randall’s plaque of patients with nephrolithiasis begins in basement membranes of thin loops of Henle.
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#CRYSTAL BUT PLUG FREE#
Epub 2014 Aug 7ĭröge W (2002) Free radicals in the physiological control of cell function. Epub 2016 Aug 17ĭaudon M, Bazin D, Letavernier E (2015) Randall’s plaque as the origin of calcium oxalate kidney stones. Epub 2015 May 29Ĭhung J, Granja I, Taylor MG, Mpourmpakis G, Asplin JR, Rimer JD (2016) Molecular modifiers reveal a mechanism of pathological crystal growth inhibition. Ĭheungpasitporn W, Rossetti S, Friend K, Erickson SB, Lieske JC (2016) Treatment effect, adherence, and safety of high fluid intake for the prevention of incident and recurrent kidney stones: a systematic review and meta-analysis.
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Ĭao LC, Honeyman TW, Cooney R, Kennington L, Scheid CR, Jonassen JA (2004) Mitochondrial dysfunction is a primary event in renal cell oxalate toxicity. J Urol 155(3):839–843Ĭao LC, Honeyman T, Jonassen J, Scheid C (2000) Oxalate-induced ceramide accumulation in Madin-Darby canine kidney and LLC-PK1 cells. īorghi L, Meschi T, Amato F, Briganti A, Novarini A, Giannini A (1996) Urinary volume, water and recurrences in idiopathic calcium nephrolithiasis: a 5-year randomized prospective study. (17)52619-1īoonla C, Wunsuwan R, Tungsanga K, Tosukhowong P (2007) Urinary 8-hydroxydeoxyguanosine is elevated in patients with nephrolithiasis. īaggio B, Gambaro G, Ossi E, Favaro S, Borsatti A (1983) Increased urinary excretion of renal enzymes in idiopathic calcium oxalate nephrolithiasis. Īsplin JR, Mandel NS, Coe FL (1996) Evidence of calcium phosphate supersaturation in the loop of Henle.
#CRYSTAL BUT PLUG UPDATE#
Īlelign T, Petros B (2018) Kidney stone disease: an update on current concepts. Īlamani BG, Rimer JD (2017) Molecular modifiers of kidney stones. KeywordsĪggarwal KP, Narula S, Kakkar M, Tandon C (2013) Nephrolithiasis: molecular mechanism of renal stone formation and the critical role played by modulators. The present writeup is an attempt to review these proposed mechanisms and strategies to interrupt crystal to calculus formation with an emphasis on potential areas of future research. Various mechanisms and hypotheses for stone formation have been described.
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The exact pathophysiology of stone formation still remains elusive. Lot of basic and clinical research on preventive strategies has been performed during the past few decades with minimal translation into routine clinical practice. However, the progress of preventive management of urolithiasis has not occurred at the same pace. There has been a significant advancement in the endourological management of urolithiasis owing to improved optics, innovation in flexible ureteroscopy and stone ablation technologies as well. Urolithiasis is one of the most important diseases in urology accounting for a significant health burden across the world.